Endocannabinoid-dependent disinhibition of orexinergic neurons: Electrophysiological evidence in leptin-knockout obese mice

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Endocannabinoid-dependent disinhibition of orexinergic neurons: Electrophysiological evidence in leptin-knockout obese mice

OBJECTIVES In the ob/ob mouse model of obesity, chronic absence of leptin causes a significant increase of orexin (OX) production by hypothalamic neurons and excessive food intake. The altered OX level is linked to a dramatic increase of the inhibitory innervation of OX producing neurons (OX neurons) and the over expression of the endocannabinoid 2-arachidonoylglycerol (2-AG) by OX neurons of o...

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The levels of the endocannabinoids anandamide and 2-arachidonoylglycerol (2-AG) are under the negative control of leptin in the rodent hypothalamus. As leptin and endocannabinoids play opposite roles in the control of reproduction, we have investigated whether the impaired fertility typical of leptin-defective ob/ob mice is due, in part, to enhanced uterine endocannabinoid levels. We found that...

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Correction: Behavioral and Electrophysiological Characterization of Dyt1 Heterozygous Knockout Mice

The following information is missing from the Funding section: This study was supported by the Bachmann-Strauss Dystonia and Parkinson Foundation, Inc. The complete, correct funding information is as follows: This work was supported by Tyler’s Hope for a Dystonia Cure, Inc., Dystonia Medical Research Foundation, Bachmann-Strauss Dystonia and Parkinson Foundation, Inc., National Institutes of He...

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ژورنال

عنوان ژورنال: Molecular Metabolism

سال: 2017

ISSN: 2212-8778

DOI: 10.1016/j.molmet.2017.04.005